Increased Protein Modification Causes Heart Disease in Friedreich’s Ataxia Patients, Study Shows
An abnormal increase in protein acetylation leads to heart disease in Friedreich’s ataxia (FA) patients and may represent a new therapeutic target for early intervention, a study finds. The study “Progressive mitochondrial protein lysine acetylation and heart failure in a model of Friedreich’s ataxia cardiomyopathy,” appeared in Plos One. The authors found a close…