Endoplasmic Reticulum Stress Could Be Treatment Target for Friedreich’s Ataxia, Study Suggests

Endoplasmic reticulum stress due to loss of frataxin protein plays a major role in the development of Friedreich’s ataxia and could be targeted for therapy, a study involving fruit flies shows. The study, “Mitofusin-Dependent ER Stress Triggers Glial Dysfunction and Nervous System Degeneration in a Drosophila Model…